Aging acceleration-2

We continue to explore how CA aging can be controlled. In the previous experiment CA aging was controlled by CA product. In the present experiment, aging of CA-3 controls aging of CA-1 and CA-2. We start with three CA and set max age  = 130. Aging acceleration of a  cell in a particular position in CA-1 is proportional to the aging of a CA-3 cell in the same position.  In CA-2 aging acceleration of the same cell is negatively proportional to the aging of the respective CA-3 cell. Which may be expressed as follows:

CA-1[aging acceleration, cell position] =   k * CA-3[aging, cell position]
CA-2[aging acceleration, cell position] = - k * CA-3[aging, cell position]
k; proportionality constant.

The arrows in the image below portray the direction of  control. After  the experiment starts, the structures of CA-1 and CA-2 change, while CA-3 remains unchanged.  Aging acceleration  in CA-1 and CA-2 is expressed in relation with  that of CA-3.   CA-3 acceleration in each position is a-0[position] (in short a-0).

The next image portrays aging of the mid cell (position = 50). Initially the age of all three mid cells is the same. Now  CA-1 ages more rapidly, and CA-2 more slowly,  than CA-3. At t = 30 CA-1 mid cell hits age max and its age is set to zero (white). CA-2 mid cell ages slowly and does not hit the max age level. After t = 30 CA-3 mid cell stops aging, and will not affect aging acceleration of  CA-1 and CA-2. Since other  cells behave differently they continue controlling CA-1 and CA-2 aging acceleration.

Below are three aging acceleration profiles. Each point represents aging acceleration of a CA-1 cell  relative to that of a CA-3 cell at  the same position.  White cells indicate that the aging of a CA-1 cell is faster than the respective CA-3 cell.  Gray colored cells age at the same velocity in CA-1 and CA-3. Black cells age slower.

Although aging of CA-1 is accelerated by CA-3, the outcome is not clear cut. Other factors also control aging, like the relationship between max age and aging velocity, or the aging of the neighbors of a particular cell.  Take the CA-3 mid cell depicted above. It stops aging altogether, yet the other keep on aging. The lowest profile expresses relative aging of CA-1 and CA-2. As expected its white points predominate. Yet in a different setting this may not be so. This is what non-linearity is about. Instead of guessing what such a profile might be, let CA compute  it for you.

nca=3; zygote -> effect[no 1000]; go109]; restoreparams; toaccel = 1;  age[[no]] +=  a[[no]];  age[[1]] +=(toaccel  a[[3]]);   age[[2]] -=(toaccel  a[[3]]);  effect[no, 130];  go[100];

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